The solution to the scarcity of islets for transplantation is to get the islet cells from living donors. This is according to the new report in Clinical Transplantation's new study. Samples of these cells obtained from living donors showed a 94% viability for transplantation against 42% from the dead donors. Clinical Transplantation is a journal for those who care for people requiring tissue or organ transplant.
The author of the study, Dr. Kwang-Won Kim said that the only known treatment for the diabetics who are dependent on insulin is transplantation of islet cells. The trouble is there is not enough supply from dead donors especially sometimes they need two such donors to obtain adequate cells to treat just one diabetic. Living donors will solve this problem because islet cells weakens right after death.
There is a downside to this because the procedure to obtain islet cells from live donors is not free from risk. There is a chance for the donors themselves to develop diabetes and who wants that? There is therefore a need for more research on this issue in order to make certain the donors will be safe and at the same time meet the demand for islet cells.
Last year UK's Department of Health announced funding from the government for a new type 1 diabetes treatment. It will spend 2.34 million euros the first year and increase this to a maximum of 7.32 million euros as it meets the demand for the service.
Previously, this type of service was offered to twelve patients in England which were funded from charitable organizations but chiefly by Diabetes UK. The new funding from the government will allow around 20 transplants in six centers that will be ready 24/7 to receive pancreas from donors and prepare the islet cells for transplantation. This will further expand to 80 transplants a year.
Won't it be grand if we had this option in the US as well.? I've tried to look around for this so that I can broadcast it from my website to give my readers the option to study and see if this fits their treatment plan. I did the same thing in Canada but the best I could find there was the pioneering islet transplantation in Edmonton, Alberta that paved the way to the current success.
Friday, May 22, 2009
Friday, May 15, 2009
Diabetes Study on Dog Owners with Type 1 Diabetes
Diabetes study has been funded by Diabetes UK to research anecdotal records on the dogs' reaction to the changes of their owners' blood sugar levels. Researchers asked type 1 diabetics who are also dog owners to participate in the study. I am revisiting this because of my interest for the diabetics to get earlier help before the hypoglycemic episode sets in.
The researchers from University of London and Queen's University in Belfast were going to investigate whether the dogs could detect the change in their owners' blood sugar level. They were therefore looking for 100 people to take part in this study.
Dr. Deborah Wells of Queen's University said they knew of anecdotal records that show dogs that were able to notice a drop in their owners' blood sugar level. The next issue of Balance was going to feature Dr. Wells' research. It would be the highlight of that issue. I looked all over the internet but couldn't find it. Maybe you did? Let me know then at this page
where you can join for free to receive alert and tips. There at the bottom of the page, you'll also see a contact form.
Those who wanted to be part of this study would have to fill out a questionnaire by mail or through the internet. Their response to the questionnaire would be confidential. They did not have to submit their names nor any personal matter. Check out the website of the Queen's University Belfast.
I found the results of another study where 138 dog owners reported that their dog showed a behavioral reaction to at least one event of hypoglycemia. Those who reported their dog reaction to the same episode to 11 or more events are in the 31.9% group.
It looked like too that their dog's age, sex, length of ownership and breed did not play a role to the response. Around 36% of owners claim that their dogs reacted when their blood sugar level went low before the owners noticed that they were exhibiting signs of low blood glucose level.
How did these dogs react? Well the responses were varied. Around 49.2% licked them, 61.5% vocalized, 30.4% jumped on them, 40.6$ nuzzled them and 41.3% stared at them intently. There was smaller proportion of dogs responding by hyperventilating, trembling or running away from the owners.
What conclusion can we derive from these reactions? It can be said that the reaction to the hypoglycemic events of the type 1 diabetic owners of these dogs happen to dogs that are not trained. This is important as who knows what kind of reaction will these dogs show after they are trained? Let us see what is the result if not of this but of another diabetes study.
The researchers from University of London and Queen's University in Belfast were going to investigate whether the dogs could detect the change in their owners' blood sugar level. They were therefore looking for 100 people to take part in this study.
Dr. Deborah Wells of Queen's University said they knew of anecdotal records that show dogs that were able to notice a drop in their owners' blood sugar level. The next issue of Balance was going to feature Dr. Wells' research. It would be the highlight of that issue. I looked all over the internet but couldn't find it. Maybe you did? Let me know then at this page
where you can join for free to receive alert and tips. There at the bottom of the page, you'll also see a contact form.
Those who wanted to be part of this study would have to fill out a questionnaire by mail or through the internet. Their response to the questionnaire would be confidential. They did not have to submit their names nor any personal matter. Check out the website of the Queen's University Belfast.
I found the results of another study where 138 dog owners reported that their dog showed a behavioral reaction to at least one event of hypoglycemia. Those who reported their dog reaction to the same episode to 11 or more events are in the 31.9% group.
It looked like too that their dog's age, sex, length of ownership and breed did not play a role to the response. Around 36% of owners claim that their dogs reacted when their blood sugar level went low before the owners noticed that they were exhibiting signs of low blood glucose level.
How did these dogs react? Well the responses were varied. Around 49.2% licked them, 61.5% vocalized, 30.4% jumped on them, 40.6$ nuzzled them and 41.3% stared at them intently. There was smaller proportion of dogs responding by hyperventilating, trembling or running away from the owners.
What conclusion can we derive from these reactions? It can be said that the reaction to the hypoglycemic events of the type 1 diabetic owners of these dogs happen to dogs that are not trained. This is important as who knows what kind of reaction will these dogs show after they are trained? Let us see what is the result if not of this but of another diabetes study.
Friday, May 8, 2009
Insulin Resistance Cause Discovered
This new discovery could pave the way to the development of new treatment to beat type 2 diabetes. Researchers at the UCSD (University of California in San Diego) School of Medicine have discovered the cause of insulin resistance.
They say that inflammation aggravated by the immune cells cause insulin resistance which leads to type 2 diabetes. Type 2 diabetes is linked to obesity which is the world’s most common metabolic disease. The theory for sometime now is that the inflammation related to obesity leads to insulin resistance.
Now with the use of mouse models, the scientists at UCSD provided evidence that by stopping inflammatory pathway, resistance to insulin and type 2 diabetes can be avoided. I am revisiting this issue hoping that they have been successful at finding treatment based on this discovery.
The lead investigators of this study are Michael Karin, PhD who is pharmacology professor at UCSD and Jerrold Olefsky, who is the Scientific Affairs’ Associate Dean and also a renowned professor of medicine. Their findings are featured in the Cell Metabolism‘s November 7 issue.
Olefsky said that their research demonstrates that insulin resistance can be disconnected from fatty fat increase that is linked to obesity. When immune cells get into fat, they release a substance called cytokines. These cause fat cells to become resistant to insulin which could lead to type 2 diabetes.
The UCSD research team demonstrated that by knocking out the component responsible for making the fat cells resistant to insulin, they were able to interrupt insulin resistance. They also used a control group of mice. Both groups of mice were fed with high fat diet. The control group diet resulted in obesity and inflammation. This in turn caused insulin resistance and type 2 diabetes.
The other group whose component that led to inflammation was knocked out also became obese. But herein lies the importance of this research. This group did not show insulin resistance at all. This led the scientists to conclude that disarming the inflammatory pathway could break off the surge that leads to insulin resistance and ultimately to type 2 diabetes.
They say that inflammation aggravated by the immune cells cause insulin resistance which leads to type 2 diabetes. Type 2 diabetes is linked to obesity which is the world’s most common metabolic disease. The theory for sometime now is that the inflammation related to obesity leads to insulin resistance.
Now with the use of mouse models, the scientists at UCSD provided evidence that by stopping inflammatory pathway, resistance to insulin and type 2 diabetes can be avoided. I am revisiting this issue hoping that they have been successful at finding treatment based on this discovery.
The lead investigators of this study are Michael Karin, PhD who is pharmacology professor at UCSD and Jerrold Olefsky, who is the Scientific Affairs’ Associate Dean and also a renowned professor of medicine. Their findings are featured in the Cell Metabolism‘s November 7 issue.
Olefsky said that their research demonstrates that insulin resistance can be disconnected from fatty fat increase that is linked to obesity. When immune cells get into fat, they release a substance called cytokines. These cause fat cells to become resistant to insulin which could lead to type 2 diabetes.
The UCSD research team demonstrated that by knocking out the component responsible for making the fat cells resistant to insulin, they were able to interrupt insulin resistance. They also used a control group of mice. Both groups of mice were fed with high fat diet. The control group diet resulted in obesity and inflammation. This in turn caused insulin resistance and type 2 diabetes.
The other group whose component that led to inflammation was knocked out also became obese. But herein lies the importance of this research. This group did not show insulin resistance at all. This led the scientists to conclude that disarming the inflammatory pathway could break off the surge that leads to insulin resistance and ultimately to type 2 diabetes.
Friday, May 1, 2009
Double Use of Diabetes Drugs
Diabetes drugs have been in use but between 2002 and 2005, girls in the range of 10 and 14 years old showed a 166% increase in the use of type 2 diabetes medications. They say the reason is obesity which is linked to type 2 diabetes. This was reported at the annual meeting of the American Public Health Association.
The reporters were the researchers from the School of Medicine of Saint Louis University and pharmacy manager Express Scripts. They also examined patterns for cholesterol, asthma, blood pressure and depression medications.
Emily R. Cox, PhD, the senior director of Express Scripts said that in the four years they studied there was an increase use of medication. Males between 15 and 19 years old increased their blood pressure medication by 15.4% while the females of the same age group decreased their use of antihypertensives by 1.6%.
It was different though for the same females with regards to taking an anti-depressant because this increased by 6.8%. All in all, the patterns show the changing prescribing actions by physicians, the rising risk factors for chronic conditions like prescribing type 2 anti diabetics, antihyperlipidemics, more office visits and therefore screening rates especially for females. The trend is for the greater use of drugs as the preferred way to treat children with these chronic conditions.
Newer research work has shown the increase in medications prescribed for children that were usually given to adults who suffer from chronic conditions. And now more and more children are given the same medicines to treat cholesterol, high blood pressure and diabetes.
This should wake all of us up into getting into the habit of eating healthy. The children are so used to eating pastries and donuts that there is really no other way but to get fat. And where did they learn this from? Of course, they learned that from us. It is our job therefore to reverse that habit and learn to choose healthier fare among the diabetes-food-list.
The researchers said this may be due the improved awareness of these conditions being present in children including the increased risk of the factors related to heart problems due to the epidemic of obesity. It looks like that about 20% of adolescents and children are obese or overweight.
The reporters were the researchers from the School of Medicine of Saint Louis University and pharmacy manager Express Scripts. They also examined patterns for cholesterol, asthma, blood pressure and depression medications.
Emily R. Cox, PhD, the senior director of Express Scripts said that in the four years they studied there was an increase use of medication. Males between 15 and 19 years old increased their blood pressure medication by 15.4% while the females of the same age group decreased their use of antihypertensives by 1.6%.
It was different though for the same females with regards to taking an anti-depressant because this increased by 6.8%. All in all, the patterns show the changing prescribing actions by physicians, the rising risk factors for chronic conditions like prescribing type 2 anti diabetics, antihyperlipidemics, more office visits and therefore screening rates especially for females. The trend is for the greater use of drugs as the preferred way to treat children with these chronic conditions.
Newer research work has shown the increase in medications prescribed for children that were usually given to adults who suffer from chronic conditions. And now more and more children are given the same medicines to treat cholesterol, high blood pressure and diabetes.
This should wake all of us up into getting into the habit of eating healthy. The children are so used to eating pastries and donuts that there is really no other way but to get fat. And where did they learn this from? Of course, they learned that from us. It is our job therefore to reverse that habit and learn to choose healthier fare among the diabetes-food-list.
The researchers said this may be due the improved awareness of these conditions being present in children including the increased risk of the factors related to heart problems due to the epidemic of obesity. It looks like that about 20% of adolescents and children are obese or overweight.
Friday, April 24, 2009
Complex Carbohydrates and Avoiding Sugar - Still the Health Protectors?
The following is an article we wrote yesterday and submitted to article directories:
Complex carbohydrates are touted to protect one from a number of diseases and conditions including cancer, cardiovascular diseases and type 2 diabetes. So knowing that we have these weapons to protect ourselves, why do some people not still see the light?
North Americans’ intake of refined sugar is estimated at 33 kg plus 20 kg more of syrup high in fructose per person each year. Fructose is used to sweeten syrups, low-fat food and beverages. The trouble with taking too much of this is that the liver changes too much sugar in the diet into lipid. The increased lipid closes down a gene namely, sex hormone binding globulin (SHBG).
This shutting down of the SHBG reduces the quantity of SHBG protein in the blood. This is not good as this protein plays an important part in regulating the quantity of estrogen and testosterone. With the less amount of SHBG comes higher risk for a number of conditions like uterine cancer.
The reduced SHBG amount also affects the metabolic state of the liver. Thus SHBG levels are employed to show a person’s risk of developing cardiovascular disease and type 2 diabetes. It is therefore good to follow the advice to avoid eating sugar and to eat complex carbohydrates.
This leads us to the question as to what complex carbohydrates we can eat. Basically, these are foods in the form of whole grain like brown rice, muesli, oats and wholegrain breads. These complex carbohydrates are broken down more gradually than those of their cousins, the simple carbohydrates. That is why they are able to provide a slow but steady source of energy during the day.
Some more examples of complex carbohydrates are bran, maize, peas, pasta, macaroni and spaghetti. These are really starch or sugar that are bonded together forming a chain so the digestive enzymes have a harder job of breaking them down making them longer to digest. This has also an added bonus in the sense that it restricts the quantity of sugar that is converted into fat.
Complex carbohydrates should be a main part of the diet. In fact it should form half of the calories each day like the aforementioned foods plus cereals, fruits and vegetables. Simple sugar should only form a few of the calories allotted each day. This is some kind of tip you will get at Free Diabetes Alert when you sign up for free newsletter.
Dr. Hammond who is scientific director of Vancouver, Canada’s Child and Family Research Institute said that SHBG can be used to monitor the functioning of the liver way before the symptoms appear. This is good news because quite a number of people who have diabetes do not even know they have it. This puts them at a disadvantage because they cannot work on strategies to beat diabetes and stop the complications.
The new findings can also be used to find out whether the drugs and dietary strategies are effective in treating the conditions. This also dismisses the assumption that SHBG is reduced by too much insulin. Insulin therefore is not to blame for the low SHBG levels but rather the sugar metabolism in the liver is what counts. So what is the moral of the story? Eat complex carbohydrates.
Complex carbohydrates are touted to protect one from a number of diseases and conditions including cancer, cardiovascular diseases and type 2 diabetes. So knowing that we have these weapons to protect ourselves, why do some people not still see the light?
North Americans’ intake of refined sugar is estimated at 33 kg plus 20 kg more of syrup high in fructose per person each year. Fructose is used to sweeten syrups, low-fat food and beverages. The trouble with taking too much of this is that the liver changes too much sugar in the diet into lipid. The increased lipid closes down a gene namely, sex hormone binding globulin (SHBG).
This shutting down of the SHBG reduces the quantity of SHBG protein in the blood. This is not good as this protein plays an important part in regulating the quantity of estrogen and testosterone. With the less amount of SHBG comes higher risk for a number of conditions like uterine cancer.
The reduced SHBG amount also affects the metabolic state of the liver. Thus SHBG levels are employed to show a person’s risk of developing cardiovascular disease and type 2 diabetes. It is therefore good to follow the advice to avoid eating sugar and to eat complex carbohydrates.
This leads us to the question as to what complex carbohydrates we can eat. Basically, these are foods in the form of whole grain like brown rice, muesli, oats and wholegrain breads. These complex carbohydrates are broken down more gradually than those of their cousins, the simple carbohydrates. That is why they are able to provide a slow but steady source of energy during the day.
Some more examples of complex carbohydrates are bran, maize, peas, pasta, macaroni and spaghetti. These are really starch or sugar that are bonded together forming a chain so the digestive enzymes have a harder job of breaking them down making them longer to digest. This has also an added bonus in the sense that it restricts the quantity of sugar that is converted into fat.
Complex carbohydrates should be a main part of the diet. In fact it should form half of the calories each day like the aforementioned foods plus cereals, fruits and vegetables. Simple sugar should only form a few of the calories allotted each day. This is some kind of tip you will get at Free Diabetes Alert when you sign up for free newsletter.
Dr. Hammond who is scientific director of Vancouver, Canada’s Child and Family Research Institute said that SHBG can be used to monitor the functioning of the liver way before the symptoms appear. This is good news because quite a number of people who have diabetes do not even know they have it. This puts them at a disadvantage because they cannot work on strategies to beat diabetes and stop the complications.
The new findings can also be used to find out whether the drugs and dietary strategies are effective in treating the conditions. This also dismisses the assumption that SHBG is reduced by too much insulin. Insulin therefore is not to blame for the low SHBG levels but rather the sugar metabolism in the liver is what counts. So what is the moral of the story? Eat complex carbohydrates.
Saturday, April 18, 2009
Body-Weight Regulation Experts Report on Research Related to Obesity
Obesity makes body parts begin to fail when the body is overloaded with extra nutrients. This is when obesity increases the risk for developing such diseases as heart problems and diabetes. Because so many parts are affected and studies focus on a particular organ at a time, what’s occurring throughout the body is overlooked.
There is an article in the November issue of Science entitled “An Integrative View of Obesity” that recommends a wider approach should be employed to find out the causes and effects of obesity. The authors are Dr. Francis Kim, Dr. Michael Schwartz and Dr. Brent E. Wisse, all of the University of Washington.
These authors investigated related studies on this issue especially the one by the Harvard University School of Public Health’s Dr. Gordon S. Gokhan S. Hotamisligil. These researches reported on what happens when the body consumes more energy in food than what it needs. This fact triggers harmful responses in body cells.
The Science article points out similarities in how different cells respond to extra nutrients. This might show why many people put on too much weight and then are not able to lose it. The authors said that the body protects its stores of fat.
Obesity they said does not happen because of the extra fat but through the defense of the raised level of fat and this should be taken into account when trying to find out why people get fat. This might also lead to the understanding why animals on low-calorie diet live longer.
It is good to know that scientists are starting to see the pattern of the cell response to extra nutrients. Among this is the way the cell responds by producing plenty of molecules that create trouble. This can harm the structures of the cells and also inhibits the last stages of protein production. The end result of this is inflammation.
This inflammation can stop insulin action and as this progresses so does the inflammation making it hard to break the cycle. Rodents fed with a diet rich in energy show impaired reaction to insulin and other signals to control the appetite.
Their brains reveal inflammation and resistance to insulin. This may make the brains unable to see they are gaining fat reserves with their brains unable to control their appetite. On the contrary, they seem hungry and keep consuming more food. It is good to see alert like this so we can act accordingly so see if you want to join us for free to see such diabetes alerts once a month.
Extra nutrients also causes inflammation to the pancreas and the authors could not help but wonder if this sets the onset for type 2 diabetes. The investigators stressed the fact this is not the only explanation for obesity and its link to diseases like heart problems and diabetes but it does show how different troubles in a variety of organs may be due to cellular reaction that is common to other parts of the body.
It is therefore imperative that an integrative approach be employed in studying the diseases that occur in tissues and cells in the whole body rather than studying just one body part or system at a time. This approach could lead to better prevention and treatment of obesity.
There is an article in the November issue of Science entitled “An Integrative View of Obesity” that recommends a wider approach should be employed to find out the causes and effects of obesity. The authors are Dr. Francis Kim, Dr. Michael Schwartz and Dr. Brent E. Wisse, all of the University of Washington.
These authors investigated related studies on this issue especially the one by the Harvard University School of Public Health’s Dr. Gordon S. Gokhan S. Hotamisligil. These researches reported on what happens when the body consumes more energy in food than what it needs. This fact triggers harmful responses in body cells.
The Science article points out similarities in how different cells respond to extra nutrients. This might show why many people put on too much weight and then are not able to lose it. The authors said that the body protects its stores of fat.
Obesity they said does not happen because of the extra fat but through the defense of the raised level of fat and this should be taken into account when trying to find out why people get fat. This might also lead to the understanding why animals on low-calorie diet live longer.
It is good to know that scientists are starting to see the pattern of the cell response to extra nutrients. Among this is the way the cell responds by producing plenty of molecules that create trouble. This can harm the structures of the cells and also inhibits the last stages of protein production. The end result of this is inflammation.
This inflammation can stop insulin action and as this progresses so does the inflammation making it hard to break the cycle. Rodents fed with a diet rich in energy show impaired reaction to insulin and other signals to control the appetite.
Their brains reveal inflammation and resistance to insulin. This may make the brains unable to see they are gaining fat reserves with their brains unable to control their appetite. On the contrary, they seem hungry and keep consuming more food. It is good to see alert like this so we can act accordingly so see if you want to join us for free to see such diabetes alerts once a month.
Extra nutrients also causes inflammation to the pancreas and the authors could not help but wonder if this sets the onset for type 2 diabetes. The investigators stressed the fact this is not the only explanation for obesity and its link to diseases like heart problems and diabetes but it does show how different troubles in a variety of organs may be due to cellular reaction that is common to other parts of the body.
It is therefore imperative that an integrative approach be employed in studying the diseases that occur in tissues and cells in the whole body rather than studying just one body part or system at a time. This approach could lead to better prevention and treatment of obesity.
Friday, April 10, 2009
Telemedicine Shows Potential Benefit for Treating Eye Diseases
Telemedicine may play an important role in diagnosing eye diseases and increasing access to excellent care and compliance of patient. This is the topic of the two studies being reported at 2007 Annual Meeting of the American Academy of Ophthalmology.
The first study looked at patients who had retinal examination before the telemedicine remote imaging system was installed. They also looked at diabetics who had retinal examination two years after the same device was installed.
Telemedicine is not new. In fact it has been noted as technically possible but this study revealed the true impact of telemedicine on the diabetics who ended up having diabetic retinopathy. Ophthalmology assistant professor Ingrid Zimmer-Galler, M.D. of John Hopkins School of Medicine’s Wilmer Eye Institute said that the system considerably increased the number of yearly retinal assessments.
Out of 1257 diabetics during the first year only 15% had the annual retinal exam. Two years after the device was installed, 71% of 1395 diabetics had the retinal exam. It should be noted that of the increase, 66% was done by a local ophthalmologist and only 33% was the result of the assessment by remote. This is indeed very encouraging as it means more diabetics are aware of the need for the annual retinal exam.
This increased awareness is vital because diabetic retinopathy affects about 5.3 million people in the US, among whom one-third do not know they have the disease. Since diabetics are twenty-five more times likely to become blind than their counterpart without the disease, early discovery and treatment will help avoid the loss of vision.
The second study involved the use of telemedicine and ophthalmology for premature babies. Columbia University College of Physicians and Surgeons’ Michael Chiang, M.D. who is also assistant professor of biomedical informatics and ophthalmology reported that the birth of premature babies is rising around the world.
The doctors gathered info on 206 eyes of 67 premature babies and performed two sets of exams. One was through ophthalmoscopic exam and the other through capturing retinal images of the same babies. After four to twelve months, the two results were compared and 86% showed the results were the same.
You can read more about this at the site below but find out first if you want to join us or not for free to receive news alert about once a month on diabetes or get them in the website as well with this free newsletter. Then you can find out more regarding Facts About Diabetes on eye complications in the Diabetes Complications Section of the website or in the sitemap.
The first study looked at patients who had retinal examination before the telemedicine remote imaging system was installed. They also looked at diabetics who had retinal examination two years after the same device was installed.
Telemedicine is not new. In fact it has been noted as technically possible but this study revealed the true impact of telemedicine on the diabetics who ended up having diabetic retinopathy. Ophthalmology assistant professor Ingrid Zimmer-Galler, M.D. of John Hopkins School of Medicine’s Wilmer Eye Institute said that the system considerably increased the number of yearly retinal assessments.
Out of 1257 diabetics during the first year only 15% had the annual retinal exam. Two years after the device was installed, 71% of 1395 diabetics had the retinal exam. It should be noted that of the increase, 66% was done by a local ophthalmologist and only 33% was the result of the assessment by remote. This is indeed very encouraging as it means more diabetics are aware of the need for the annual retinal exam.
This increased awareness is vital because diabetic retinopathy affects about 5.3 million people in the US, among whom one-third do not know they have the disease. Since diabetics are twenty-five more times likely to become blind than their counterpart without the disease, early discovery and treatment will help avoid the loss of vision.
The second study involved the use of telemedicine and ophthalmology for premature babies. Columbia University College of Physicians and Surgeons’ Michael Chiang, M.D. who is also assistant professor of biomedical informatics and ophthalmology reported that the birth of premature babies is rising around the world.
The doctors gathered info on 206 eyes of 67 premature babies and performed two sets of exams. One was through ophthalmoscopic exam and the other through capturing retinal images of the same babies. After four to twelve months, the two results were compared and 86% showed the results were the same.
You can read more about this at the site below but find out first if you want to join us or not for free to receive news alert about once a month on diabetes or get them in the website as well with this free newsletter. Then you can find out more regarding Facts About Diabetes on eye complications in the Diabetes Complications Section of the website or in the sitemap.
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